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yet blocks the agonist-induced conformational changes in TM6
YL-109 179 yet blocks the agonist-induced conformationalYL 109 has ability to inhibit breast cancer cell growth and invasiveness in vitro and in vivo. YL 109 repressed the sphere forming ability and the expression of stem cell markers in MDA MB 231 mammosphere cultures. YL 109 increased the expression of carboxyl terminus of Hsp70 interacting protein (CHIP), which suppresses tumorigenic and metastatic potential of breast cancer cells by inhibiting the oncogenic pathway. YL 109 induced CHIP transcription
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